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|a 1349567444
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|a QH430
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|a 576.5
|2 23
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|a Advances in genetics.
|n Volume 110 /
|c editors, Stanley Maloy, Kelly Hughes.
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|a Cambridge, MA :
|b Academic Press,
|c 2022.
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|a 1 online resource (1 volume)
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|a text
|b txt
|2 rdacontent
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|a computer
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|2 rdamedia
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|a online resource
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|a Print version record.
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|t Genetic factors governing bacterial virulence and host plant susceptibility during Agrobacterium infection /
|r Benoit Lacroix and Vitaly Citovsky --
|t Epigenetic regulation of angiogenesis in tumor progression /
|r Vanessa Desantis, Antonio G. Solimando, and Domenico Ribatti.
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|a Intro -- Advances in Genetics -- Copyright -- Contents -- Contributors -- Chapter One: RecBCD enzyme and Chi recombination hotspots as determinants of self vs. non-self: Myths and mechanisms -- 1. Introduction -- 2. Chi is more frequent in many bacterial species and E. coli phages and plasmids than in E. coli itself -- 3. Chi is active in ``foreign�� species -- 4. Is Chi ``over-represented�� in E. colis genome? -- 4.1. Nucleotides are not randomly arranged in the E. coli genome -- 4.2. Amino acids are not quite randomly associated in E. coli proteins -- 4.3. Codon usage is even more non-random than amino acid usage in E. coli -- 4.4. Preferential codon usage can account for Chi�s high frequency in E. coli -- 5. ``Chi�� sequence of S. aureus also comports with its codon usage -- 6. Temperate phage P1 contains 50 Chi sites, likely uses them to its advantage, and appears to select for Chi as a recomb ... -- 7. A myth: ``Chi converts RecBCD from phage destruction to DNA repair�� -- 8. An observation: RecBCD destroys DNA in E. coli only if it cannot recombine -- 9. Mechanisms employing RecBCD that do distinguish self vs. non-self DNA -- 10. Conclusion: Telling self from non-self is complex -- 11. Methods of data analysis -- Acknowledgments -- References -- Chapter Two: Multiple regulatory mechanisms for pH homeostasis in the gastric pathogen, Helicobacter pylori -- 1. Introduction -- 1.1. Two mechanisms of acid-resistance -- 1.2. Urease activity needs to be regulated -- 2. H. pylori reference genome, gene nomenclature and genomic diversity -- 3. Urease function and urea supply -- 4. Regulation of urease activities -- 4.1. Sensors of pH changes -- 4.2. Effectors controlling the expression of the urease gene cluster -- 4.3. Nickel homeostasis and urease activation -- 4.3.1. Nickel importing and exporting.
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|a 4.3.2. Nickel sequestrating and storage proteins -- 4.3.3. Different pathways for nickel insertion between urease and hydrogenase -- 4.3.4. Regulatory proteins for maintaining nickel homeostasis -- Acknowledgments -- References.
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|a Genetics.
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|a G�en�etique.
|0 (CaQQLa)201-0069234
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|a genetics.
|2 aat
|0 (CStmoGRI)aat300138822
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650 |
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|a Genetics
|2 fast
|0 (OCoLC)fst00940117
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700 |
1 |
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|a Maloy, Stanley R.,
|e editor.
|1 https://isni.org/isni/0000000120207840
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700 |
1 |
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|a Hughes, Kelly Thomas,
|e editor.
|1 https://isni.org/isni/0000000117673787
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776 |
0 |
8 |
|i Print version:
|t Advances in genetics. Volume 110.
|d Amsterdam : Academic Press, 2022
|z 9780443188787
|w (OCoLC)1346472810
|
856 |
4 |
0 |
|u https://sciencedirect.uam.elogim.com/science/bookseries/00652660/110
|z Texto completo
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