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Pathophysiology of nonalcoholic steatohepatitis /

Detalles Bibliográficos
Clasificación:Libro Electrónico
Autor principal: Basaranoglu, Metin
Formato: Electrónico eBook
Idioma:Inglés
Publicado: New York : Nova Biomedical Books, ©2009.
Temas:
Acceso en línea:Texto completo

MARC

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100 1 |a Basaranoglu, Metin. 
245 1 0 |a Pathophysiology of nonalcoholic steatohepatitis /  |c Metin Basaranoglu and Brent A. Neuschwander-Tetri. 
260 |a New York :  |b Nova Biomedical Books,  |c ©2009. 
300 |a 1 online resource (viii, 112 pages) :  |b illustrations 
336 |a text  |b txt  |2 rdacontent 
337 |a computer  |b c  |2 rdamedia 
338 |a online resource  |b cr  |2 rdacarrier 
504 |a Includes bibliographical references (pages 60-97) and index. 
588 0 |a Print version record. 
505 0 |a PATHOPHYSIOLOGY OFNONALCOHOLICSTEATOHEPATITIS -- NOTICE TO THE READER -- CONTENTS -- PREFACE -- ABBREVIATIONS -- PATHOPHYSIOLOGY OF EXCESSIVE FATACCUMULATION IN THE LIVER IN THEABSENCE OF ALCOHOL ABUSE: NAFLD -- OBESITY, INSULIN RESISTANCE ANDHYPERINSULINEMIA AS RISK FACTORS FOR NAFLD -- SOURCES OF LIVER FAT -- Dysregulated Peripheral Lipolysis -- Hepatic de Novo Lipogenesis (DNL) -- Uptake of Dietary Fat into the Liver -- FATES OF LIVER FAT -- Very-low-density Lipoprotein (VLDL) Synthesis and Secretion -- Mitochondrial β-Oxidation 
505 8 |a Peroxisomal Fatty Acid β-OxidationCytochrome P450 Fatty Acid Ï? (Omega)-Oxidation -- Local and Generalized Inflammation in NAFLD -- NASH: THE PATHOGENESIS OFHEPATOCELLULAR INJURY IN NAFLD -- ROLE OF ANIMAL MODELS IN UNDERSTANDING THEPATHOGENESIS OF NASH -- The ob/ob Mouse -- The Methionine and Choline Deficient (MCD) Diet -- Other Dietary Models -- Transition from Simple Steatosis and NASH to NASH-AssociatedHCC: A new Murine NASH-Associated Hepatic Neoplasia Mo -- OXIDATIVE STRESS AND THE PATHOGENESIS OFNASH -- Mitochondria as a Source of Oxidant Stress 
505 8 |a P450 as a Source of Oxidant StressIron, Oxidant Stress and NASH -- MITOCHONDRIAL DYSFUNCTION AND ATPDEPLETION -- FREE FATTY ACID TOXICITY -- ENDOGENOUS TOXINS:ENDOTOXIN AND GUT-DERIVED ETHANOL -- ADIPOCYTOKINES -- Leptin -- Adiponectin -- TNF-Î" -- IL-6 -- Resistin -- Regulation of Hepatic Immunity and Increased Sensitivity toHepatocellular Injury -- HEPATOCYTE APOPTOSIS IN NAFLD -- HEPATIC FIBROGENESIS IN NASH -- ROLE OF STELLATE CELLS AND CYTOKINES INHEPATIC FIBROGENESIS -- HEPATOCELLULAR CARCI -- PATHOPHYSIOLOGY OF THEPATHOLOGICAL FEATURES OF NASH 
505 8 |a MICROVESICULAR AND MACROVESICULAR STEATOSISINFLAMMATION -- GLYCOGEN NUCLEI -- LIPOGRANULOMAS -- HEPATOCELLULAR BALLOONI -- HEPATOCELLULAR BALLOONING -- MALLORY BODIES AND STRESS PROTEINS -- GENETIC SUSCEPTIBILITY TO NASH AND THE BASISOF NASH-PATHOPHYSIOLOGY -- NASH Prevalence in Different Racial and Ethnic Groups -- NAFLD and Genes Associated with Lipid and Glucose Metabolism, Oxidant and Anti-Oxidant Systems, and ProinflammatoryCytokines -- SUMMARY -- REFERENCES -- INDEX 
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650 0 |a Liver  |x Diseases. 
650 0 |a Fatty liver. 
650 1 2 |a Fatty Liver  |x physiopathology 
650 2 |a Fatty Liver 
650 6 |a Stéatose hépatique. 
650 7 |a MEDICAL  |x Hepatology.  |2 bisacsh 
650 7 |a Fatty liver  |2 fast 
650 7 |a Liver  |x Diseases  |2 fast 
758 |i has work:  |a Pathophysiology of nonalcoholic steatohepatitis (Text)  |1 https://id.oclc.org/worldcat/entity/E39PCGtBBjykTckfWwXpGtqgfC  |4 https://id.oclc.org/worldcat/ontology/hasWork 
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