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The Na+, K+ Pumps Keep Us Going /

Detalles Bibliográficos
Autor principal: Clausen, Torben (Autor)
Autor Corporativo: ProQuest (Firm)
Formato: Electrónico eBook
Idioma:Inglés
Publicado: Aarhus, Denmark : Aarhus University Press, 2016.
Colección:Book collections on Project MUSE.
Temas:
Acceso en línea:Texto completo

MARC

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020 |z 9788771249392 
035 |a (OCoLC)1108538592 
040 |a MdBmJHUP  |c MdBmJHUP 
100 1 |a Clausen, Torben,  |e author. 
245 1 4 |a The Na+, K+ Pumps Keep Us Going /   |c by Torben Clausen. 
264 1 |a Aarhus, Denmark :  |b Aarhus University Press,  |c 2016. 
264 3 |a Baltimore, Md. :  |b Project MUSE,   |c 2023 
264 4 |c ©2016. 
300 |a 1 online resource (120 pages):   |b illustrations (some color), charts (some color. 
336 |a text  |b txt  |2 rdacontent 
337 |a computer  |b c  |2 rdamedia 
338 |a online resource  |b cr  |2 rdacarrier 
505 0 |a The Na+, K+ pump and its discovery -- The transport and distribution of Na+ and K+ in skeletal muscle and how they are quantified -- Rapid passive fluxes of Na+ and K+ start muscle contraction and set a limit to excitability -- The definition, structure and function of the Na+, K+-pump -- The synthesis and localization of the Na+, K+ pumps in skeletal muscle -- Measurements of the content of [3H]ouabain binding sites indicate that in skeletal muscle, the major part of these binding sites of the Na+, K+ pumps are localized on the outer surface of sarcolemma and in the t-tubules -- The content of Na+, K+ pumps in skeletal muscles and how it can be quantified -- Muscle contractions, Na+, K+ transport and sodium potassium fatigue -- The rate of [³H]ouabain binding to sarcolemma can be quantified and what information may this provide? -- Measurements of [3H]ouabain binding to other cell types and preparations -- Acute and long-term regulation of Na+, K+ pumps in skeletal muscle -- Catecholamines and caffeine -- Peptide hormones stimulating the Na+, K+ pumps -- Insulin-like growth Factore-I (IGF-I) -- CGRP and other calcitonins -- Amylin, related peptides, and other stimuli for the Na+, K+ -pumps -- The importance of Na+-influx in causing depolarization and muscle fatigue -- Effect of varying Na+, K+-pump stimulating agents on intracellular Na+ in rat soleus muscles and how this may compensate functional defects caused by plasma membrane leakage -- Long-Term regulation of Na+, K+ pump content -- Training, inactivity and denervation -- Muscular dystrophy and McArdle disease -- Hyperkalemic periodic paralysis in horses, human subjects and mutant mice with a similar geneti anomaly -- K+ deficiency and K+ restoration -- Thyroid hormones ans starvation -- Thermogenic actions of thyroid hormones and malignant hyperthermia (MH) -- Diabetes -- Steroid hormones, glucocorticoids, aldosterone -- Major conclusions and general perspectives. 
588 |a Description based on print version record. 
650 7 |a Sodium/potassium ATPase.  |2 fast  |0 (OCoLC)fst01124101 
650 7 |a Sodium  |x Physiological transport.  |2 fast  |0 (OCoLC)fst01124035 
650 7 |a Potassium  |x Physiological transport.  |2 fast  |0 (OCoLC)fst01073302 
650 6 |a Potassium  |x Transport physiologique. 
650 6 |a Sodium  |x Transport physiologique. 
650 6 |a Pompe à sodium. 
650 0 |a Potassium  |x Physiological transport. 
650 0 |a Sodium  |x Physiological transport. 
650 0 |a Sodium/potassium ATPase. 
655 7 |a Electronic books.   |2 local 
710 2 |a ProQuest (Firm) 
710 2 |a Project Muse.  |e distributor 
830 0 |a Book collections on Project MUSE. 
856 4 0 |z Texto completo  |u https://projectmuse.uam.elogim.com/book/110386/ 
945 |a Project MUSE - Custom Collection 
945 |a Project MUSE - 2023 Annual Backfile - Unpurchased