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LEADER |
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SCIDIR_ocn968251440 |
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OCoLC |
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20231120112206.0 |
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170113s2017 enk ob 001 0 eng d |
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|b eng
|e rda
|e pn
|c OPELS
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|d YDX
|d OCLCO
|d OCLCQ
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|d D6H
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|d S2H
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|d REDDC
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019 |
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|a 1105170588
|a 1105575258
|a 1229063281
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|a 9780128094273
|q (e-book)
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|a 0128094273
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|z 9780128054208
|q (print)
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|z 0128054204
|q (print)
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|a (OCoLC)968251440
|z (OCoLC)1105170588
|z (OCoLC)1105575258
|z (OCoLC)1229063281
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|a QH671
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|a QU 375
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082 |
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4 |
|a 571.9/36
|2 23
|
245 |
0 |
0 |
|a Autophagy :
|b cancer, other pathologies, inflammation, immunity, infection and aging.
|n Volume 11 /
|c edited by M.A. Hayat.
|
264 |
|
1 |
|a London, United Kingdom :
|b Academic Press, an imprint of Elsevier,
|c 2017.
|
300 |
|
|
|a 1 online resource
|
336 |
|
|
|a text
|b txt
|2 rdacontent
|
337 |
|
|
|a computer
|b c
|2 rdamedia
|
338 |
|
|
|a online resource
|b cr
|2 rdacarrier
|
500 |
|
|
|a Includes index.
|
588 |
0 |
|
|a Online resource; title from PDF title page (ScienceDirect, viewed January 13, 2017).
|
504 |
|
|
|a Includes bibliographical references at the end of each chapters and index.
|
505 |
8 |
0 |
|g Part II:
|t Role in disease:
|g 9.
|t Methods for measuring autophagy levels in disease /
|r Kanchan Phadwal, Dominic Kurian --
|g 10.
|t Regulation of the DNA damage response by autophagy /
|r Vinay V. Eapen, David P. Waterman, Brenda Lemos, James E. Haber --
|g 11.
|t Autophagy and cancer /
|r Yoshitaka Isaka, Tomonori Kimura, Yoshitsugu Takabatake, Atsushi Takahashi --
|g 12.
|t ULK1 can suppress or promote tumor growth under different conditions /
|r Tian Mao, Ouyang Liang --
|g 13.
|t X-box-binding protein 1 splicing induces an autophagic response in endothelial cells : molecular mechanisms in ECs and atherosclerosis /
|r Sophia Kelaini, Rachel Caines, Lingfang Zeng, Andriana Margariti --
|g 14.
|t Small molecule-mediated simultaneous induction of apoptosis and autophagy /
|r Sudhakar Jinka, Rajkumar Banerjee --
|g 15.
|t Intestinal autophagy defends against Salmonella infection /
|r Thomas A. Parker, Kailiang Jia --
|g 16.
|t Autophagy and LC3-associated phagocytosis mediate the innate immune response /
|r Larissa D. Cunha, Jennifer Martinez.
|
520 |
|
|
|a 'Autophagy: Cancer, Other Pathologies, Inflammation, Immunity, Infection, and Aging' is an eleven volume series that discusses in detail all aspects of autophagy machinery in the context of health, cancer, and other pathologies. Autophagy maintains homeostasis during starvation or stress conditions by balancing the synthesis of cellular components and their deregulation by autophagy. This series discusses the characterization of autophagosome-enriched vaccines and its efficacy in cancer immunotherapy. Autophagy serves to maintain healthy cells, tissues, and organs, but also promotes cancer survival and growth of established tumors. Impaired or deregulated autophagy can also contribute to disease pathogenesis. Understanding the importance and necessity of the role of autophagy in health and disease is vital for the studies of cancer, aging, neurodegeneration, immunology, and infectious diseases. Comprehensive and forward-thinking, these books offer a valuable guide to cellular processes while also inciting researchers to explore their potentially important connections. --
|c Provided by publisher.
|
650 |
|
0 |
|a Autophagic vacuoles.
|
650 |
|
0 |
|a Cell death.
|
650 |
|
0 |
|a Cytology.
|
650 |
|
2 |
|a Autophagy
|0 (DNLM)D001343
|
650 |
|
2 |
|a Cell Biology
|0 (DNLM)D003585
|
650 |
|
2 |
|a Cytology
|0 (DNLM)D000093222
|
650 |
|
6 |
|a Cellules
|x Mort.
|0 (CaQQLa)201-0110794
|
650 |
|
6 |
|a Cytologie.
|0 (CaQQLa)201-0040099
|
650 |
|
6 |
|a Autophagie (Cytologie)
|0 (CaQQLa)201-0273011
|
650 |
|
7 |
|a SCIENCE
|x Life Sciences
|x Anatomy & Physiology.
|2 bisacsh
|
650 |
|
7 |
|a Autophagic vacuoles
|2 fast
|0 (OCoLC)fst00824188
|
650 |
|
7 |
|a Cell death
|2 fast
|0 (OCoLC)fst00850180
|
650 |
|
7 |
|a Cytology
|2 fast
|0 (OCoLC)fst00886282
|
700 |
1 |
|
|a Hayat, M. A.,
|d 1940-
|e editor.
|
856 |
4 |
0 |
|u https://sciencedirect.uam.elogim.com/science/book/9780128054208
|z Texto completo
|