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The Antigens /
| Clasificación: | Libro Electrónico |
|---|---|
| Otros Autores: | |
| Formato: | Electrónico eBook |
| Idioma: | Inglés |
| Publicado: |
San Diego ; London :
Academic Press,
1987.
|
| Temas: | |
| Acceso en línea: | Texto completo |
Tabla de Contenidos:
- Front Cover; The Antigens; Copyright Page; Table of Contents; List of Contributors; Preface; Contents of Other Volumes; CHAPTER 1. Ir Genes: Antigen-Specific GeneticRegulation of the Immune Response; I. Introduction/Definitions; II. Developing Concepts; III. The Role of Genetic Restriction of Cell Interactionsin Ir Gene Function; IV. Levels of Imposition of Genetic Restriction; Role of the Thymic Environment; Studies in Chimeras; V. la Antigens as Ir Gene Products; VI. Non-MHC-Linked Ir Genes; VII. The Role of Suppressor Cells in Ir Gene Control; VIII. The T Cell Repertoire Model.
- IX. The Antigen Presentation ModelX. T Cell Influences on Antibody Specificity; XI. Epilogue; Acknowledgments; References; CHAPTER2. Molecular Genetics of Class II (la) Antigens; I. Introduction; II. Immunogenetics; III. Genomic Organization of the I Region; IV. Correlation of Immunogeneticand Molecular Genetic Maps; VI. Polymorphism of Class II Antigens; VII. Correlation between Structure and Functionof Class II Antigens; VIII. Regulation of la Expression; IX. Conclusion; Acknowledgments; References; CHAPTER3. Antigen-Specific T Cell Clones and T Cell Factors; I. Introduction.
- II. Functional T Cell Hybridomas and Their SolubleProductsIII. Antigen-Specific Virus-Transformed FunctionalT Cell Lines; IV. T Cell Continuous Lines and Clones; V. The T Cell Receptor; VI. Concluding Remarks; References; CHAPTER4. Infection and Autoimmunity; I. Introduction; II. Molecular Mimicry between Microbialand Host Antigens; III. Autoantibodies Produced by Patients with Infections; IV. Altered Self; V. Polyclonal Activation; VI. The Role of Infection in the Expression of HLAClass II Antigens in Self Tissues; VII. The Anti-Idiotypic Network; VIII. Specific Therapy of Autoimmune Disease.