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Oxidative damage to plants : antioxidant networks and signaling /
With contributions that review research on this topic throughout the world, Oxidative Damage to Plants covers key areas of discovery, from the generation of reactive oxygen species (ROSs), their mechanisms, quenching of these ROSs through enzymatic and non-enzymatic antioxidants, and detailed aspect...
| Clasificación: | Libro Electrónico |
|---|---|
| Otros Autores: | |
| Formato: | Electrónico eBook |
| Idioma: | Inglés |
| Publicado: |
Amsterdam :
Academic Press,
2014.
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| Temas: | |
| Acceso en línea: | Texto completo |
Tabla de Contenidos:
- Front Cover; Oxidative Damage to Plants; Copyright Page; Dedication; Contents; Foreword; Preface; Acknowledgments; List of Contributors; 1 Reactive Oxygen Species and Photosynthesis; 1.1 Introduction; 1.2 Photosynthesis: Light Reactions; 1.2.1 Photosystems; 1.2.2 Light-Absorbing Antenna Systems; 1.2.2.1 Structure of Antenna Systems; 1.2.3 Photosystem II Structure and Function; 1.2.3.1 Water Oxidation and O2 Evolution; 1.2.4 Electron Flow through the PS II and Cyt b6f Complex; 1.2.4.1 Q Cycle; 1.2.5 PS I Reaction Center and Reduction of NADP+; 1.2.5.1 ATP Synthesis.
- 1.3 Photosynthesis: Carbon reactions and Photorespiration1.3.1 The Calvin Cycle; 1.3.1.1 Light-regulated Enzymes in Calvin Cycle; 1.3.2 Photorespiration; 1.4 Reactive Oxygen Species (ROS); 1.4.1 Chemical Forms of ROS; 1.4.2 Varied Sources of ROS in Plant Cells; 1.4.2.1 Chloroplasts; 1.4.2.1.1 Singlet Oxygen; 1.4.2.1.2 Production of 1O2 in PS I; 1.4.2.1.3 Superoxide Radical; 1.4.2.1.4 Hydrogen Peroxide; 1.4.2.1.5 Hydroxyl Radicals; 1.4.2.2 Other Organelles; 1.4.2.2.1 Endoplasmic Reticulum, Peroxisomes and Glyoxysomes; 1.4.2.2.2 Plasmamembrane and the Apoplast Compartment.
- 1.4.3 Effect of Environmental Factors on ROS Production during Photosynthesis1.5 Protection of Photosynthetic Plants against Ros; 1.5.1 Protection from Excess Excitation Energy: Photoprotection; 1.5.1.1 Nonphotochemical Quenching; 1.5.1.1.1 Flexible versus Sustained Energy Dissipation; 1.5.1.1.2 Lutein-related Thermal Dissipation; 1.5.1.1.3 Thermal Dissipation in Desiccation-tolerant Plants; 1.5.1.2 Quenching by Inactive PS II Reaction Centers; 1.5.1.3 State I/State II Transition; 1.5.1.4 Aggregation of LHCII; 1.5.1.5 Cyclic Electron Flow around PS II and PS I.
- 1.5.1.5.1 Cyclic Electron Flow around PS II1.5.1.5.2 Cyclic Electron Flow around PS I; 1.5.1.6 Water-Water Cycle; 1.5.1.7 Chlororespiration and Photorespiration; 1.5.1.8 Reaction Center Quenching; 1.5.2 Protection by Scavenging Systems: Antioxidative Defense; 1.5.2.1 Enzymes; 1.5.2.1.1 Superoxide Dismutase (EC 1.15.1.1); 1.5.2.1.2 Catalase (EC 1.11.1.6); 1.5.2.1.3 Peroxidase (EC 1.11.1.7); 1.5.2.1.4 Ascorbate Peroxidase (EC 1.11.1.11); 1.5.2.1.5 Monodehydroascorbate Reductase (EC 1.6.5.4); 1.5.2.1.6 Dehydroascorbate Reductase (EC 1.8.5.1); 1.5.2.1.7 Glutathione Reductase (EC 1.6.4.2)
- 1.5.2.1.8 Glutathione Peroxidase (EC 1.11.1.9)1.5.2.2 Metabolites; 1.5.2.2.1 Ascorbic Acid; 1.5.2.2.2 Glutathione; 1.5.2.2.3 Tocopherols; 1.5.2.2.4 Carotenoids; 1.5.2.2.5 Phenolic Compounds; 1.5.2.2.6 Differential Intercellular Partitioning of Antioxidants in C4 Plans; 1.6 Redox and ROS Signaling; 1.6.1 Redox Signaling through PQ; 1.6.2 Redox Signaling and NPQ; 1.6.3 Chlorophyll Synthesis and Redox Signaling; 1.6.4 Redox Signaling and Dithiol/Disulphide Exchanges; 1.6.5 ROS Signaling; 1.6.6 Involvement of Kinases in the Perception Mechanisms of EEE and Redox Signaling.