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Platelets and aspirin-induced asthma : pathogenesis and melatonin /

Platelets and Aspirin-Induced Asthma is the first book to be published that reflects research conducted on aspirin-induced asthma pathogenesis. It is examined through positions of neuroimmunoendocrine interactions in organism. The Melatonin hormone plays a key role, being the regulator and coordinat...

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Detalles Bibliográficos
Clasificación:Libro Electrónico
Autor principal: Evsyukova, Helen V.
Otros Autores: Joseph, M. (Editor )
Formato: Electrónico eBook
Idioma:Inglés
Publicado: Amsterdam : Elsevier/Academic Press, 2014.
Temas:
Acceso en línea:Texto completo
Tabla de Contenidos:
  • Front Cover; Platelets and Aspirin-Induced Asthma; Copyright Page; Contents; List of Abbreviations; About the Author; Foreword; Introduction; 1 Clinical Picture of Aspirin-Induced Asthma (AIA); 1.1 Clinical Risk Factors and Course of Aspirin-Induced Asthma; 1.2 Peculiarities of Pulmonary Function, Laboratory Data, and Pulmonary Circulation State in AIA Patients; 2 Diffuse Neuroimmunoendocrine System (DNIES) and its Role in the Human Body; 2.1 Definition; 2.2 Melatonin (MT) as DNIES Hormone and its Role in Regulating the Activity of the Organism's Functional Systems.
  • 2.2.1 MT Synthesis and Metabolism in the Organism2.2.2 The Role of MT in Regulation of Biological Rhythms; 2.2.3 MT as an Endogenous Antioxidant; 2.2.4 Role of MT in Regulation of Platelet-Vessel Hemostasis; 2.2.5 MT's Effect on Nitric Oxide Production in Organism; 2.2.6 The Role of MT in the Regulation of Arachidonic Acid (AA) Metabolism; 3 State of DNIES in Aspirin-Induced Asthma; 3.1 MT Production in Patients with AIA; 3.2 Platelet-Vessel Hemostasis in Patients with AIA; 3.2.1 Platelet Functional Activity in Patients with AIA.
  • 3.2.2 Peculiarities of Nitric Oxide,Thromboxane and Prostacyclin Production in AIA3.3 Adrenal Glucocorticoid's Function in Patients with AIA; 3.4 Peculiarities of Immune System in Patients with AIA; 3.5 AA Metabolism in Patients with AIA; 4 Pathogenesis of Aspirin-Induced Asthma; Conclusion; References.