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Gene expression in the central nervous system /
Gene expression is an active ongoing process that maintains a functional CNS, as proteins are being made on a continual basis. Processes such as learning and memory, nerve cell repair and regeneration and its response to stress are critically dependent on gene expression. This volume highlights the...
| Clasificación: | Libro Electrónico |
|---|---|
| Autores Corporativos: | , , , , |
| Otros Autores: | |
| Formato: | Electrónico Congresos, conferencias eBook |
| Idioma: | Inglés |
| Publicado: |
Amsterdam ; New York :
Elsevier,
1995.
|
| Colección: | Progress in brain research ;
v. 105. |
| Temas: | |
| Acceso en línea: | Texto completo Texto completo |
MARC
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| 111 | 2 | |a Stanford International Neuroscience Symposium |n (2nd : |d 1993 : |c Beijing, China) | |
| 245 | 1 | 0 | |a Gene expression in the central nervous system / |c edited by Albert C.H. Yu [and five others]. |
| 264 | 1 | |a Amsterdam ; |a New York : |b Elsevier, |c 1995. | |
| 300 | |a 1 online resource (xvi, 341 pages) : |b illustrations (some color). | ||
| 336 | |a text |b txt |2 rdacontent | ||
| 337 | |a computer |b c |2 rdamedia | ||
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| 490 | 1 | |a Progress in brain research, |x 0079-6123 ; |v volume 105 | |
| 500 | |a Papers from the Second Stanford International Neuroscience Symposium held in Beijing, China, October 19-22, 1993, organized by the Department of Pathology at Stanford University School of Medicine and sponsored by the School of Medicine, Stanford University, Beijing Medical University and the Faculty of Medicine, Chinese University of Hong Kong. | ||
| 504 | |a Includes bibliographical references and index. | ||
| 520 | |a Gene expression is an active ongoing process that maintains a functional CNS, as proteins are being made on a continual basis. Processes such as learning and memory, nerve cell repair and regeneration and its response to stress are critically dependent on gene expression. This volume highlights the role of gene expression in normal CNS function, and presents many research methods at the cutting edge of neuroscience, which will provide insight into therapeutic approaches through which the control of gene expression may be used in the treatment of many nervous system diseases. | ||
| 588 | 0 | |a Print version record. | |
| 506 | |a Online full text access is restricted to licensed organisations. | ||
| 505 | 0 | |a Front Cover; Gene Experssion in the Central Nervous System; Copyright Page; List of Contributors; Preface; Contents; Section I: Techniques in Analysis of Gene Expression; Chapter 1. The gene knockout technology for the analysis of learning and memory, and neural development; Chapter 2. Molecular biology of transmissible spongiform encephalopathies; Chapter 3. Molecular genetic analyses of myelin deficiency and cerebellar ataxia; Chapter 4. Gene expression of serotonergic neurons in the central nervous system: molecular and developmental analysis | |
| 505 | 8 | |a Section II: Signal Transduction and Gene ExpressionChapter 5. The involvement of PKC and multifunctional CaM Kinase II of the post-synaptic neuron in induction and maintenance of long-term potentiation; Chapter 6. Neuronal calcium channels encoded by the a1A subunit and their contribution to excitatory synaptic transmission in the CNS; Chapter 7. Synaptic vesicle proteins and regulated exocytosis; Chapter 8. The molecular organization of voltage-dependent K+ channels in vivo; Chapter 9. Decoding Ca2+ signals to the nucleus by multifunctional CaM kinase | |
| 505 | 8 | |a Chapter 10. Kainate-induced changes in gene expression in the rat hippocampusChapter 11. Mechanisms of neuronal plasticity as analyzed at the single cell level; Section III: Development, Differentiation, and Aging; Chapter 12. Plasticity and commitment in the developing cerebral cortex; Chapter 13. Growth factors in the CNS and their effects on oligodendroglia; Chapter 14. Social control of cell size: males and females are different; Chapter 15. The differentiation and function of the touch receptor neurons of Caenorhabditis elegans | |
| 505 | 8 | |a Chapter 16. Functions of the L2/HNK-1 carbohydrate in the nervous systemChapter 17. Neurotrophic factors and their receptors; Chapter 18. Induction of non-catalytic TrkB neurotrophin receptors during lesion- induced synaptic rearrangement in the adult rat hippocampus; Chapter 19. Plasticity of developing neuromuscular synapses; Section IV: Diseases and Injury; Chapter 20. A RT-PCR study of gene expression in a mechanical injury model; Chapter 21.Stimulation of phospholipase A2 expression in rat cultured astrocytes by LPS, TNFa and IL-1� | |
| 505 | 8 | |a Chapter 22. Correlation between proto-oncogene, fibroblast growth factor and adaptive response in brain infarctChapter 23. Gene expression in astrocytes during and after ischemia; Chapter 24. Gene expression of neurotropic retrovirus in the CNS; Chapter 25. Cholecystokinin octapeptide (CCK-8): a negative feedback control mechanism for opioid analgesia; Chapter 26. The transport of neurotransmitters into synaptic vesicles; Chapter 27. Preliminary molecular neurobiology study on the pathogenesis of primary epilepsy | |
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| 650 | 0 | |a Neurogenetics |v Congresses. | |
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| 700 | 1 | |a Yu, Albert C. H., |e editor. | |
| 710 | 2 | |a Stanford University. |b Department of Pathology, |e organizer of meeting. | |
| 710 | 2 | |a Stanford University. |b School of Medicine, |e sponsoring body. | |
| 710 | 2 | |a Beijing yi ke da xue, |e sponsoring body. | |
| 710 | 2 | |a Chinese University of Hong Kong. |b Faculty of Medicine, |e sponsoring body. | |
| 776 | 0 | 8 | |i Print version: |t Gene expression in the central nervous system. |d Amsterdam ; New York : Elsevier, 1995 |w (DLC) 95021940 |w (OCoLC)32823756 |
| 830 | 0 | |a Progress in brain research ; |v v. 105. | |
| 856 | 4 | 0 | |u https://sciencedirect.uam.elogim.com/science/bookseries/00796123/105 |z Texto completo |
| 856 | 4 | 0 | |u https://sciencedirect.uam.elogim.com/science/book/9780444818522 |z Texto completo |
| 856 | 4 | |u https://sciencedirect.uam.elogim.com/science/bookseries/00796123/105 |z Texto completo | |