Endocrine problems in cancer : molecular basis and clinical management /

Endocrine Problems in Cancer.

Detalles Bibliográficos
Clasificación:Libro Electrónico
Otros Autores: Jung, Roland T., Sikora, Karol
Formato: Electrónico eBook
Idioma:Inglés
Publicado: London : Heinemann Medical Books, 1984.
Temas:
Cancer > Endocrine aspects.
Endocrine glands > Diseases.
Endocrine System Diseases > etiology
Metabolic Diseases > etiology
Molecular Biology
Neoplasms > complications
Endocrine System Diseases
Cancer > Aspect endocrinien.
Glandes endocrines > Maladies.
Cancer patients > Endocrine system > Diseases
Acceso en línea:Texto completo
Tabla de Contenidos:
  • Front Cover; Endocrine Problems in Cancer: Molecular Basis and Clinical Management; Copyright Page; Table of Contents; List of Contributors; Preface; Chapter 1. Molecular Biology of Cancer; INTRODUCTION; TECHNIQUES OF MOLECULAR BIOLOGY; DIFFERENTIATION; CANCER CELLS; CELL COMMUNICATION; PRODUCTION OF HORMONES BY CANCER CELLS; REFERENCES; Chapter 2. Endocrine Markers in Cancer; INTRODUCTION; MECHANISMS OF HUMORAL SYNDROMES; CONCLUSION; REFERENCES; Chapter 3. Genetic Problems of Endocrine Significance; INTRODUCTION; MULTIPLE ENDOCRINE NEOPLASIA TYPE I (MEN I; WERNER SYNDROME)
  • MULTIPLE ENDOCRINE NEOPLASIA TYPE II (MEN II SIPPLE'S SYNDROME); PHAEOCHROMOCYTOMA; ADRENAL CORTICAL TUMOURS; PITUITARY; THYROID (EXCLUDING MEDULLARY CARCINOMA); PARATHYROID; GONADAL DYSGENESIS; TESTICULAR FEMINISATION; SYNDROMES WITH CONSTITUTIONAL CHROMOSOMAL ABNORMALITY; SCREENING OF FAMILY MEMBERS AT RISK; ACKNOWLEDGEMENTS; REFERENCES; Chapter 4. Nutritional Support; CANCER CACHEXIA-WHY DOES IT OCCUR?; DOES HYPERALIMENTATION REVERSE THE NUTRITIONAL AND METABOLIC CONSEQUENCES OF CANCER?; DOES HYPERALIMENTATION STIMULATE TUMOUR GROWTH?
  • IS THE RESPONSE TO CANCER THERAPY IMPROVED BY ADJUNCT HYPERALIMENTATION?DOES HYPERALIMENTATION REDUCE THE SIDE EFFECTS OF CANCER THERAPY?; DOES HYPERALIMENTATION HAVE A ROLE IN PATIENTS WITH CANCER?; HOW DOES ONE ASSESS THE PATIENT FOR SIGNS OF MALNUTRITION?; HOW DOES ONE DESIGN A SUITABLE NUTRITIONAL REGIMEN?; REFERENCES; Chapter 5. Glucose Homeostasis; INTRODUCTION; HYPERGLYCAEMIA; HYPOGLYCAEMIA; OTHER CAUSES OF HYPOGLYCAEMIA; SUMMARY; REFERENCES; Chapter 6. Disorders of Mineral Metabolism; INTRODUCTION; HYPERCALCAEMIA; HYPOPHOSPHAT AEMIA; BONE AND MALIGNANT DISEASE
  • CLINICAL FEATURES OF HYPERCALCAEMIA OF MALIGNANCYMEDIATORS OF HYPERCALCAEMIA IN MALIGNANCY; TUMOUR-ASSOCIATED OSTEOMALACIA; ECTOPIC CALCITONIN PRODUCTION; OTHER FACTORS INFLUENCING CALCIUM HOMEOSTASIS IN MALIGNANCY; MAGNESIUM AND CANCER; MANAGEMENT OF HYPERCALCAEMIA; REFERENCES; Chapter 7. Growth and Development; INTRODUCTION; MALIGNANT TUMOURS OF THE CENTRAL NERVOUS SYSTEM; TUMOURS WHICH MAY CAUSE IMPAIRED GROWTH; TUMOURS WHICH MAY CAUSE DELAYED PUBERTY (Table 7.2); TUMOURS WHICH MAY CAUSE PRECOCIOUS PUBERTY; TUMOURS OF OTHER ENDOCRINE GLANDS AFFECTING GROWTH AND DEVELOPMENT
  • DIAGNOSIS AND MANAGEMENT OF ENDOCRINE DISTURBANCEMANAGEMENT OF ENDOCRINE DISTURBANCE; REFERENCES; Chapter 8. Hypopituitarism; HYPOTHALAMIC LESIONS PRODUCING HYPOPITUITARISM; HYPOPITUITARISM SECONDARY TO PITUITARY NEOPLASMS; METASTATIC DISEASE OF THE PITUITARY GLAND; CARCINOMA OF BREAST; CARCINOMA OF BRONCHUS; HYPOPITUITARISM IN METASTATIC DISEASE OF THE PITUITARY GLAND; DIFFERENTIATION OF PITUITARY METASTASES FROM PITUITARY TUMOURS; INCIDENCE OF TUMOURS DEVELOPING IN HYPOPITUITARY PATIENTS; HYPOPITUITARISM-CLINICAL PICTURE; LABORATORY INVESTIGATIONS; RADIOLOGICAL ASSESSMENT; TREATMENT

Ejemplares similares