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EBSCO_ocn965196278 |
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|a UAMI
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|a Advances in medicine and biology.
|n Volume 108 /
|c Leon V. Berhardt, editor.
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264 |
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|a New York :
|b Nova Science Publishers, Inc. :
|b Nova Biomedical,
|c [2017]
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|c ©2017
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300 |
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|a 1 online resource
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|a Preface; Hyperpigmentation: Etiology, Topical Treatments and Effectiveness; Abstract; Introduction; Melanogenesis: Physiologic Aspect; Causes of Hyperpigmentation; Epidermal Melanocytosis; Epidermal Pigmented Lesions; Freckles, Lentigines and Solar Lentigo; Epidermal Acquired Pigmented Disorders; Melasma; Acanthosis Nigricans; Dermal Melanocytosis; Dermal Pigmented Lesions; Nevus of Ota and Nevus of Ito; Hori nevus; Dermal Acquired Pigmented Dermatoses; Periorbital Hyperpigmentation; Erythema Dyschromicum Perstans; Riehl Melanosis; Erythromelanosis Follicularis Faciei.
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|a Poikiloderma of CivatteInherited Pigmented Disorders; Familial Lentiginosis Syndrome; Peutz-Jeghers Syndrome; Carney Complex; LEOPARD Syndrome; PTEN Hamartoma Tumor Syndromes; Benign Acquired Lentiginosis; Laugier-Hunziker Syndrome; Post-Inflammatory Hyperpigmentation; Drug and Chemical-Induced Hyperpigmentation; Endocrine and Metabolic Disorders; Addison's Disease; Hemochromatosis; Miscellaneous; Treatment of Skin Hyperpigmentation; Depigmenting Therapies with Lightening Topical Agents; Physical Treatments; Novel Terapeutical Proposal; References; Ankylosing Spondylitis: A Review; Abstract.
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|a 1. Introduction2. Ankylosing Spondylitis; 2.1. Aetiology and Incidence; 2.2. Pathophysiology; 2.3. Diagnosis; 2.3.1. Symptoms; 2.3.2. Laboratory Tests; 2.3.3. Imaging; 2.4. Management of the Disease; Surgery; Pharmacological Treatment; Non-Steroidal Anti-Inflammatory Drugs; Anti-Tumour Necrosis Factor (TNF); Non-Pharmacological Therapy; Physical Therapy; Education; Conclusion; References; Pulmonary and Cutaneous CO2 Emission Monitoring under Anesthesia; Patients' Induced Pluripotent Stem Cells to Model Thiopurine Induced Pancreatitis.
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|a Prediction of Thiopurine-Induced Pancreatitis: A Possible Application of Patients' Induced Pluripotent Stem CellsPersonalized Medicine as a Tool to Tailor Therapy and to Prevent Adverse Thiopurine Reactions; Adverse Drug Reactions and Drug-Induced Pancreatitis; Thiopurines in the Treatment of IBD and Manifestation of Adverse Drug Reactions as Pancreatitis; Genetic Markers for Thiopurine-Induced Pancreatitis in Inflammatory Bowel Disease Patients; Induced Pluripotent Stem Cells (iPSC): A Ground-Breaking Tool for Personalized Medicine.
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|a Exocrine Pancreatic Cells from Patients' iPSC as Most Appropriate Cell Subtype to Model TIPThe Molecular Mechanism of TIP Investigated Using Patient Specific iPSC Derived Exocrine Pancreatic Cells; Biotransformation Hypothesis; Innate Immunity Hypothesis; Adaptive Immunity Hypothesis; Caveats of Exocrine Pancreatic Cells Derived from Patients' iPSC as a Model for TIP; The Prevention of TIP Using Patients' iPSC; Conclusion; Acknowledgments; Conflict of Interest; References; Bioluminescence Microscopy in Live Cells: Consideration of Experimental Factors and Practical Recommendations.
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|a Online resource; title from PDF title page (EBSCO, viewed March 2, 2017).
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|a Includes bibliographical references and index.
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