Cell proliferation : processes, regulation and disorders /

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Detalles Bibliográficos
Clasificación:Libro Electrónico
Otros Autores: Zhang, Changhong, 1972- (Editor ), Zeng, Xiangqiong, 1979- (Editor )
Formato: Electrónico eBook
Idioma:Inglés
Publicado: New York : Nova Biomedical, [2013]
Colección:Cell biology research progress.
Temas:
Cell proliferation.
Cell differentiation.
Cellules > Prolifération.
Cellules > Différenciation.
SCIENCE > Life Sciences > Developmental Biology.
Acceso en línea:Texto completo
Tabla de Contenidos:
  • CELL PROLIFERATION: PROCESSES, REGULATION AND DISORDERS; CELL PROLIFERATION: PROCESSES, REGULATION AND DISORDERS; Library of Congress Cataloging-in-Publication Data; Contents; Preface; Chapter I: Glutamate and its Receptors in Controlling Proliferation of Oligodendrocyte Progenitor Cells; Abstract; 1. Introduction; 2. OPCs Represent the Largest Pool of Proliferating Cells in the Brain; 3. Glutamate Receptors on Oligodendroglial Progenitors and their Role during Cell Proliferation in Culture; 4. Glutamatergic Synapses on Oligodendroglial Progenitors and their Possible Role during Proliferation.
  • 4.1. OPCs Receive Functional Synaptic Input from Neurons4.2. Morphology of OPCs during Mitosis; 4.3. Neuron-glial Synapses during Mitosis; 4.4. Are Neuron-glia Synapses Involved in Regulation of OPC Proliferation?; 5. Proliferation of Oligodendroglial Progenitors In Vivo and its Modulation by Electrical Activity in Axons; 5.1. Neuronal Activity Controls Proliferation of Oligodendroglial Progenitors In Vivo; 5.2. How do Electrically Active Axons Signal to Glial Cells to Divide?; Conclusion; Acknowledgments; References.
  • Chapter II: The Two Faces of TGF-ß in Breast Cancer: Tumour Suppressor and Tumour PromoterAbstract; List of Abbreviations; 1. Introduction of the TGF-ß Superfamily; 2. The Mechanism of TGF-ß Signal Transduction; 2.1. The TGF-Ý Pathway; 2.2. Non-Smad Signalling; 2.3. Regulation of the TGF-Ý-Induced Smad Pathway; 3. The Dual Role of TGF-ß in Breast Cancer; 3.1. Inhibition of Tumourigenesis; 3.2. Promotion of Tumour Progression; 3.3. EMT; 3.4. From Primary Site to Circulation; 3.5. Targeting Specific Organs; Conclusion; Acknowledgments; References.
  • Chapter III: MicroRNAs and Their Therapeutic Potential for Vascular Smooth Muscle Cell Proliferation in RestenosisAbstract; 1. Introduction; 2. SMC Proliferation in Restenosis; 3. MiRNAs in SMC Proliferation; 3.1. miR-143 and miR-145; 3.2. miR-221 and miR-222; 3.3. miR-1 and miR-133; 3.4. miR-21; 3.5. miR-26a; 3.6. miR-208a; 3.7. let-7d; 4. MiRNAs in Cell Cycle; Conclusion; References; Chapter IV: Impaired Proliferation as a Component of the Pathogenesis of Follicular Persistence Associated with Cystic Ovarian Disease; Abstract; 1. Introduction; 2. Regulation of the Cell Cycle in the Ovary.
  • 3. Ovarian Cysts4. Altered Proliferation in Follicular Cysts; 5. Decreased Apoptosis Contributes to Follicular Persistence; Conclusion; References; Chapter V: Extracellular Protein-Induced Plant Cell Proliferation; Abstract; 1. Introduction; 2. Cell Systems to Investigate Cell Proliferation; 3. Conditioned Medium Enhances Cell Division during Somatic Embryogenesis; 4. Chitinases/PR Proteins Involvement in Cell Proliferation; 5. AGP-mediated Promotion of Cell Proliferation; 6. Other Extracellular Proteins Identified during Proliferation; 7. Cell Proliferation versus Cell Differentiation.

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