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Tabla de Contenidos:
  • COAGULATION ; COAGULATION ; CONTENTS ; PREFACE; A NEW APPROACH TO THE THEORY OF BROWNIAN COAGULATION AND DIFFUSION-LIMITED REACTIONS ; Abstract ; 1. Part 1. Brownian Coagulation Theory ; 1.1. Introduction ; 1.2. Diffusion Relaxation in Ensemble of Brownian Particles ; 1.3. Coagulation Rate Equation ; 1.3.1. Applicability of the Diffusion Approach to Particles Coagulation ; 1.3.2. Diffusion Mixing Condition; 1.4. Kinetic Regime: High Collision Frequency (0 c) ; 1.5. Kinetic Regime: Low Collision Frequency (0 c) ; 1.5.1. Continuum Mode (Ra) ; 1.5.2. Free Molecular Mode (Ra).
  • 1.5.3. Transition Mode (Ra) 1.5.4. Interpolation Formulas; 1.5.5. Applicability Range of the Kinetic Approach ; 1.6. Next Approximation of the Random Walk Theory ; 1.6.1. Brownian Particles Coagulation; 1.6.2. Heavy Vapor Molecules Condensation ; 1.7. Discussion; CONCLUSION ; 2. Part 2. Diffusion-Limited Reaction Rate Theory ; 2.1. Introduction ; 2.2. Rate Equations ; 2.2.1. Applicability of the Diffusion Approach to Particles Collisions ; 2.2.2. Diffusion Mixing Condition ; 2.2.3. Applicability of the Reaction Rate Equation ; 2.3. Reaction Rate in 3-D Case.
  • 2.3.1. Continuum Mode (BAABBArrRaa,) 2.3.2. Free Molecular Mode (BAABaaR,) ; 2.4. Reaction Rate in 2-D Case ; 2.5. Reaction Rate on 3-D Discrete Lattice ; 2.6. Reaction Rate on 2-D Discrete Lattice ; CONCLUSION ; APPENDIX ; ACKNOWLEDGMENTS ; REFERENCES ; DEREGULATION OF COAGULATION DURING SEPSIS-INDUCED DISSEMINATED INTRAVASCULAR COAGULATION ; Abstract ; INTRODUCTION ; 1. Physiological Coagulation and Fibrinolysis ; 2. Overview of Sepsis ; 3. Overview of Disseminated Intravvascular Coagulation ; 4. Deregulation of Coagulation by Bacteria.
  • 5. Deregulation of Fibrinolysis by Bacteria 6. Coagulation Factor and Inhibitor Therapies ; CONCLUSION ; Authors' CONTRIBUTIONS ; ACKNOWLEDGEMENTS ; REFERENCES ; COAGULATION: KINETIC, STRUCTURE, FORMATION AND DISORDERS ; Abstract ; 1. Introduction ; 2. Kinetics of Coagulation Systems and Clot Formation ; 2.1. Primary Hemostasis ; 2.2. Coagulation Is Constituted by Interacting Elements ; 2.3. The Extrinsic Route of Coagulation ; 2.4. The Intrinsic Route of Coagulation ; 2.5. The Cellular Model of Coagulation ; 2.6. Activation of the Coagulation System; 3. Kinetics.
  • 3.1. Coagulation and Inflammatory Components 3.2. Coagulation and Interaction with Endothelial Cells and Leukocytes ; 3.3. Cross-Talk between Clotting and Complement System (C) ; 4. Disorders ; 4.1. Thrombophilia ; 4.1.1. Acquired Factors Associated to Thrombosis ; 4.1.1.1. Procoagulant States and Their Association with Humoral Immunity ; 4.1.1.2. Concepts of Antibodies in Thrombophilia ; 4.2. Genetic Factors Associated to Thrombophilia ; 4.2.1. Deficiency of Antithrombin III ; 4.2.2. Deficiency of Protein C ; 4.2.3. Deficiency of Protein S ; 4.2.4. Mutation of Factor V.