Vascular responses to pathogens /

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Vascular Responses to Pathogens focuses on the growing research from leaders in the field for both the short and long-term impact of pathogens on the vasculature. It discusses various organisms, including bacteria, parasites, and viruses, and their role in key events leading to vascular disease. For...

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Detalles Bibliográficos
Clasificación:Libro Electrónico
Otros Autores: Gavins, Felicity N. E. (Editor ), Stokes, Karen Y. (Editor )
Formato: Electrónico eBook
Idioma:Inglés
Publicado: London : Academic Press, an imprint of Elsevier, [2016]
Temas:
Pathogenic microorganisms.
Blood-vessels.
Cardiovascular system.
Vascular Diseases > immunology
Vascular Diseases > microbiology
Infections > immunology
Infections > metabolism
Blood Vessels
Cardiovascular System
Micro-organismes pathogènes.
Vaisseaux sanguins.
Appareil cardiovasculaire.
HEALTH & FITNESS > Diseases > General.
MEDICAL > Clinical Medicine.
MEDICAL > Diseases.
MEDICAL > Evidence-Based Medicine.
MEDICAL > Internal Medicine.
Blood-vessels
Cardiovascular system
Pathogenic microorganisms
Acceso en línea:Texto completo
Tabla de Contenidos:
  • Cover
  • Title Page
  • Copyright Page
  • Contents
  • List of Contributors
  • Overview
  • Chapter 1
  • Sepsis
  • Introduction
  • The microcirculation in sepsis
  • The endothelium
  • Nitric oxide and sepsis
  • Arterioles
  • Capillaries
  • Venules
  • Permeability
  • Blood cells
  • The Inflammatory Response in Sepsis
  • Platelets in sepsis
  • Thrombosis and coagulation in sepsis
  • Models
  • Genetics and sepsis
  • Treatments for sepsis
  • Conclusions
  • Acknowledgment
  • References
  • Chapter 2
  • Neutrophil-Mediated Vascular Host Defense
  • IntroductionBloodstream infections
  • Conventional vascular host defense
  • Neutrophil extracellular trap mediated vascular immunity
  • Immunothrombosis
  • NET-mediated tissue damage
  • Conclusions
  • References
  • Chapter 3
  • Platelets as Mediators of the Vascular Response to Infection
  • Introduction
  • Platelet and hemostasis
  • Platelets and the response to infection
  • Platelet-bacteria interactions
  • Direct binding
  • Indirect binding
  • Shear stress
  • Secreted products
  • Platelet activation
  • Platelets and sepsis
  • Conclusions
  • GBS Surface Adhesins and InvasinsGBS Pili and Other ECM-Binding Proteins
  • Signal-transduction mechanisms
  • Analysis of the host cell transcriptome in response to bacterial infection
  • Meningococcal Infection
  • GBS Infection
  • Meningococcal Manipulation of the Outer Cell Membrane Lipid Composition
  • Summary
  • Acknowledgments
  • References
  • Chapter 5
  • Vascular Responses to Chlamydia pneumoniae Infection
  • Introduction
  • Direct interaction with vascular cells
  • Receptor-Mediated Binding and Uptake
  • C. pneumoniae Uses the Mannose 6-Phosphate Receptor on Endothelial CellsC. pneumoniae Binds to and Activates the LOX-1 Receptor on Endothelial Cells
  • The C. pneumoniae Developmental Cycle and Persistent Infection
  • Effects on Host Cell Energy Metabolism
  • Inflammatory Activation of Vascular Cells and the Role of Toll-Like Receptors
  • Foam Cell Formation
  • Effects on Host Cell Turnover
  • Systemic responses to infection and the impact on the vasculature
  • Evidence of the Association of C. pneumoniae Infection and Atherosclerosis in Humans
  • Clinical Trials

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