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Human longevity : omega-3 fatty acids, bioenergetics, molecular biology, and evolution /
More than 7 billion people inhabit the earth and all of them are subject to aging. This book is aimed at persons interested in a molecular explanation of how our cells age. Human Longevity: Omega-3 Fatty Acids, Bioenergetics, Molecular Biology, and Evolution is built on the proposition that we age a...
| Clasificación: | Libro Electrónico |
|---|---|
| Autores principales: | , |
| Formato: | Electrónico eBook |
| Idioma: | Inglés |
| Publicado: |
Boca Raton :
CRC Press,
2014.
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| Temas: | |
| Acceso en línea: | Texto completo |
Tabla de Contenidos:
- Front Cover; Contents; Preface; Acknowledgments; About the Authors; Chapter 1: Mitochondrial Hypothesis of Aging Is Undergoing Revision; Chapter 2: Oxidative Stress Defined as a Deadly Free Radical-Mediated Chain Reaction : Case History of Paraquat; Chapter 3: Membranes of Deep-Sea Bacteria as Surrogates for Mitochondrial Membranes of Humans; Chapter 4: Protective Mechanisms for EPA Membranes in C. elegans and Their Relationship to Life Span.
- Chapter 5: Remarkable Longevity of Queens of Social Insects Likely Involves Dietary Manipulation to Minimize Levels of Polyunsaturates and Decrease Membrane PeroxidationChapter 6: Membrane Peroxidation Hypothesis Helps Explain Longevity in Birds, Rodents, and Whales; Chapter 7: Did Longevity Help Humans Become Super Humans?; Chapter 8: Mitochondrial Diseases and Aging Have Much in Common; Chapter 9: Revised Mitochondrial Hypothesis of Aging Highlights Energy Deficiency Caused by Errors of Replication (Mutations) of mtDNA; Chapter 10: Benefits of Polyunsaturated Mitochondrial Membranes.
- Chapter 11: Mitochondrial Membranes as a Source of Reactive Oxygen Species (ROS)Chapter 12: Mitochondrial Membranes as Major Targets of Oxidation; Chapter 13: Apoptosis Caused by Oxidatively Truncated Phospholipids Can Be Reversed by Several Mechanisms, Especially Enzymatic Detoxification; Chapter 14: Selective Targeting of HUFAs Away from Cardiolipin and Beta-Oxidation Combine to Protect Mitochondrial Membranes against Oxidative Damage; Chapter 15: Oxygen Limitation Protects Mitochondrial Phospholipids, Especially Cardiolipin.
- Chapter 16: Uncoupling Proteins (UCPs) of Mitochondria Purposely Waste Energy to Prevent Membrane DamageChapter 17: Mitochondrial Fission Protects against Oxidative Stress by Minting a Continuous Supply of Cardiolipin and Other Polyunsaturated Phospholipids; Chapter 18: Mitophagy Eliminates Toxic Mitochondria; Chapter 19: Longevity Genes Likely Protect Membranes; Chapter 20: Aging as a Cardiolipin Disease That Can Be Treated; Back Cover.