Acne : causes and practical management /

"Acne is a common ailment for teenagers but can persist well into middle age. Although the formation of comedones in hair follicles is quite well understood, the actual causes initiating the process are less well so. Many theories have been forwarded, from hormones through cleanliness to diet,...

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Detalles Bibliográficos
Clasificación:Libro Electrónico
Autor principal: Danby, F. William (Autor)
Formato: Electrónico eBook
Idioma:Inglés
Publicado: Chichester, West Sussex ; Hoboken, NJ : Wiley Blackwell, 2015.
Temas:
Acne.
Acne > Treatment.
Suppuration.
Sweat glands > Diseases.
Bacterial diseases.
Infection.
Diseases.
Skin > Diseases.
Rosacea.
Acne Vulgaris
Acneiform Eruptions
Hidradenitis
Suppuration
Skin Diseases, Infectious
Facial Dermatoses
Skin and Connective Tissue Diseases
Sweat Gland Diseases
Bacterial Infections
Infections
Bacterial Infections and Mycoses
Disease
Skin Diseases, Bacterial
Sebaceous Gland Diseases
Skin Diseases
Hidradenitis Suppurativa
Rosacea
Acné.
Acné > Traitement.
Éruptions acnéiformes.
Face > Dermatoses.
Glandes sudorifères > Maladies.
Maladies bactériennes.
Maladies.
Peau > Maladies.
Acné rosacée.
disease.
illness.
HEALTH & FITNESS > Diseases > General.
MEDICAL > Clinical Medicine.
MEDICAL > Diseases.
MEDICAL > Evidence-Based Medicine.
MEDICAL > Internal Medicine.
Sweat glands > Diseases
Skin > Diseases
Infection
Diseases
Bacterial diseases
Acne
Acne > Treatment
Acceso en línea:Texto completo
Tabla de Contenidos:
  • Machine generated contents note: Genetics,
  • Diet,
  • Hormones,
  • Stress,
  • Comedones (plugs in pores),
  • Blemishes-a brief catalogue,
  • Nodules,
  • Scars and sinuses,
  • Support,
  • Nomenclature,
  • The three acnes and grading,
  • Acne vulgaris,
  • Acne rosacea,
  • Acne inversa (hidradenitis suppurativa),
  • Grading the three acnes,
  • Acne vulgaris,
  • Acne rosacea,
  • Acne inversa (hidradenitis suppurativa),
  • 1.1. Acne vulgaris,
  • 1.1.1. Terminology,
  • 1.1.2. The starting point,
  • 1.2. Acne rosacea,
  • 1.2.1. The "pimply" part,
  • 1.2.2. The "redness" part,
  • 1.2.3. The third part, the firm fibrosis,
  • 1.2.4. Part four-ocular rosacea,
  • 1.2.5. Putting it all together,
  • 1.2.6. The inflammatory epiphenomena in acne rosacea,
  • 1.2.7. The "acne rosacea" versus "rosacea" controversy,
  • 1.2.8. Summary,
  • 1.3. Acne inversa (formerly hidradenitis suppurativa),
  • 1.3.1. Before the rupture, where and why?,
  • 1.3.2. After the rupture, what next?,
  • 1.3.3. So what invaders are important in acne inversa?,
  • 1.3.4. What makes this disease behave so much worse than acne vulgaris?,
  • 1.3.5. So what can one possibly do to settle down all this inflammation?,
  • 1.3.6. So how do you get rid of all this material?,
  • 1.3.7. What does the future offer?,
  • 1.4. The psychology of acne,
  • 1.4.1. Acne as a stress,
  • 1.4.2. Acne and self-image,
  • 1.4.3. Isotretinoin therapy and the psyche,
  • 1.4.4. The isotretinoin-depression question,
  • 1.4.5. Isotretinoin in perspective,
  • 2.1. Anatomy,
  • 2.2. Genetics,
  • 2.2.1. Acne vulgaris,
  • 2.2.2. Acne rosacea,
  • 2.2.3. Acne inversa/hidradenitis suppurativa (AI/HS),
  • 2.2.4. The Scottish twins,
  • 2.3. Epigenetics,
  • 2.3.1. The farmer's boys,
  • 2.4. Embryology,
  • 2.5. Histology,
  • 2.5.1. Onwards and downwards,
  • 2.5.2. What is going on inside the FPSU?,
  • 2.6. Physiology,
  • 2.6.1. Hair first,
  • 2.6.2. Oil second,
  • 2.6.3. Last but definitely not least: the follicle,
  • 2.6.4. Looking deeper,
  • 2.7. Biochemistry,
  • 2.8. Hormones, enzymes, receptors, and the intracrine system,
  • 2.8.1. The intracrine system,
  • 2.9. FoxO1 and mTORC1,
  • 2.9.1. The next step,
  • 2.9.2. The broad view,
  • 3.1. Acne vulgaris,
  • 3.2. Acne rosacea,
  • 3.3. Acne inversa/hidradenitis suppurativa (AI/HS),
  • 3.4. Other variants,
  • 3.4.1. Malassezia folliculitis,
  • 3.4.2. Eosinophilic pustular folliculitis (Ofuji's disease),
  • 3.4.3. Dissecting terminal folliculitis,
  • 3.4.4. Acne keloidalis,
  • 3.4.5. Epidermal growth factor receptor (EGFR) inhibitor eruption,
  • 3.4.6. Acne excoriee des jeunes files,
  • 4.1. The endogenous hormones,
  • 4.1.1. Androgens and their sources,
  • 4.1.2. Estrogens and their sources,
  • 4.1.3. Progesterone and the progesteroids,
  • 4.1.4. Insulin,
  • 4.1.5. Growth hormone and insulin-like growth factor-1,
  • 4.2. The exogenous hormones,
  • 4.2.1. Anabolic steroids,
  • 4.2.1.1. Mothers' milk,
  • 4.2.1.2. Muscle makers,
  • 4.2.2. Oral contraceptive hormones,
  • 4.2.2.1. Oral estrogens,
  • 4.2.2.2. Oral progestins,
  • 4.2.2.3. Extended cycles,
  • 4.2.3. Other exogenous birth control hormones,
  • 4.2.3.1. Implants,
  • 4.2.3.2. Intrauterine devices,
  • 4.2.3.3. Intravaginal devices,
  • 4.2.3.4. Topicals: the patches,
  • 4.2.3.5. Intramuscular (depot) injections,
  • 4.2.4. Dietary sources of hormones,
  • 4.2.4.1. The impact of diet on acne,
  • 4.2.4.1.1. The ice cream salesman's son,
  • 4.2.4.1.2. Reproductive hormones,
  • 4.2.4.1.3. Insulin,
  • 4.2.4.1.4. Insulin-like growth factor 1 (IGF-1),
  • 4.2.4.1.5. Growth factors and androgens combined,
  • 4.2.4.1.6. Dairy intolerance,
  • 4.2.4.2. Carbohydrate load versus dairy load,
  • 5.1. Chemicals and medications,
  • 5.2. Endocrine imitators and disruptors,
  • 5.2.1. Environmental contamination,
  • 5.3. Foods,
  • 5.3.1. Iodine and bromine,
  • 5.3.2. Chocolate,
  • 5.3.3. Casein and whey,
  • 5.4. Photodamage, glycation, and the acne and aging processes,
  • 5.5. Smoking and nicotine,
  • 6.1. Propionibacterium acnes (P. acnes),
  • 6.1.1. Normal role of P. acnes,
  • 6.1.2. Pathogenic role of P.
  • Acnes,
  • 6.2. Malassezia species,
  • 6.2.1. Normal role,
  • 6.2.2. Immunogenicity,
  • 6.2.3. Pruritogenicity,
  • 6.2.4. Malassezia in the acnes,
  • 6.3. Staph, Strep, and Gram-negative organisms,
  • 6.4. Demodex,
  • 6.5. Vellus hairs,
  • 7.1. Innate immunity,
  • 7.2. Adaptive (acquired) immunity,
  • 7.3. Inflammation as the primary acnegen,
  • 7.4. Mediators, cellular and humoral, and neuroimmunology,
  • 7.5. Allergy (shared antigens),
  • 7.6. Inflammation, pigment, and PIH,
  • 7.7. Inflammation and scarring,
  • 8.1. Prevention,
  • 8.2. General principles of management,
  • 8.3. Diet,
  • 8.3.1. Dairy,
  • 8.3.1.1. The deli-planning heiress,
  • 8.3.1.2. The pharmaceutical executive,
  • 8.3.2. Carbohydrates, glycemic load, and hyperinsulinemia,
  • 8.3.3. The paleolithic diet,
  • 8.3.4. High-fructose corn syrup (HFCS),
  • 8.3.5. Metformin,
  • 8.3.6. Synthesis and summary,
  • 8.4.Comedolytics and other topicals,
  • 8.4.1. Standard topical comedolytics,
  • 8.4.1.1. Retinoids,
  • 8.4.1.2. Benzoyl peroxide,
  • 8.4.1.3. Salicylic acid,
  • 8.4.1.4. Alpha and beta-hydroxy adds,
  • 8.4.2. Unclassified topicals,
  • 8.4.2.1. Azelaic acid,
  • 8.4.2.2. Sulfur,
  • 8.4.2.3. Zinc compounds,
  • 8.4.2.4. Resorcinol,
  • 8.4.3. Systemic comedolytics,
  • 8.4.3.1. Vitamin A,
  • 8.4.3.2. Isotretinoin,
  • 8.4.3.2.1. Teratogenicity,
  • 8.4.3.2.2. Contraception,
  • 8.4.3.2.3. Inflammatory bowel disease,
  • 8.4.3.2.4. Depression,
  • 8.4.3.2.5. Other side effects,
  • 8.4.3.2.6. The convict who looked like Chief,
  • 8.4.3.3. Acitretin,
  • 8.4.3.4. Summary,
  • 8.5. Anti-inflammatories and antimicrobials,
  • 8.5.1. Antibiotics as anti-inflammatories,
  • 8.5.1.1. In acne vulgaris,
  • 8.5.1.2. In acne rosacea,
  • 8.5.1.3. In acne inversa,
  • 8.5.1.4. In dissecting terminal folliculitis (DTP) and acne keloidalis,
  • 8.5.2. Antibiotics as antibiotics,
  • 8.5.3. Ketoconazole, ivermectin, and crotamiton,
  • 8.5.3.1. In acne vulgaris,
  • 8.5.3.2. In acne rosacea,
  • 8.5.3.3. In acne inversa/hidradenitis suppurativa and dissecting folliculitis and cellulitis,
  • 8.5.4. Steroids,
  • 8.5.4.1. The Marine,
  • 8.5.5. Nonsteroidal anti-inflammatory drugs (NSAIDs) and biologics,
  • 8.5.6. Phototherapy,
  • 8.5.7. Post-inflammatory hyperpigmentation,
  • 8.5.7.1. Prognosis,
  • 8.6. Hormone manipulations and therapy,
  • 8.6.1. Birth control pill selection,
  • 8.6.1.1. Estrogens,
  • 8.6.1.1.1. Warnings,
  • 8.6.1.2. Progestins,
  • 8.6.2. Androgen receptor blockade,
  • 8.6.2.1. Spironolactone,
  • 8.6.2.2. Cyproterone acetate,
  • 8.6.2.3. Flutamide,
  • 8.6.2.4. Drospirenone,
  • 8.6.2.5. Topical androgen blockers,
  • 8.6.3. Dihydrotestosterone minimization,
  • 8.6.3.1. Finasteride,
  • 8.6.3.2. Dutasteride,
  • 8.6.3.3. Diet,
  • 8.6.4. Phototherapy-hormone interactions,
  • 8.7. Surgery,
  • 8.7.1. Acne vulgaris,
  • 8.7.1.1. Acne surgery for patients,
  • 8.7.1.2. Acne surgery for physicians,
  • 8.7.2. Acne rosacea,
  • 8.7.3. Acne inversa/hidradenitis suppurativa,
  • 8.7.3.1. Mini-unroofing by punch biopsy,
  • 8.7.3.2. Unroofing,
  • 8.7.3.2.1. The Trucker,
  • 8.7.3.3. Wide surgical excision,
  • 8.7.3.4. Healing options,
  • 8.7.3.4.1. Primary closure,
  • 8.7.3.4.2. Secondary intention,
  • 8.7.3.4.3. Split-thickness mesh grafting,
  • 8.8. Lights and lasers,
  • 8.8.1. Light and other radiation in acne,
  • 8.8.1.1. Radiation's targets,
  • 8.8.1.2. Light as a practical acne therapy,
  • 8.8.2. Lasers,
  • 9.1. Epidemiology,
  • 9.2. Pathogenesis,
  • 9.3. Team up with Mother Nature,
  • 9.4. Targeting therapy,
  • 9.4.1. Clinical manifestations,
  • 9.4.2. Pathology,
  • 9.4.3. Diagnostic evaluation,
  • 9.4.4. Overview and general approach to treatment,
  • 9.4.5. Milk and pregnancy,
  • 9.4.6. Active therapy,
  • 9.4.6.1. Avoidance of harm,
  • 9.4.6.2. Lesion-directed therapy,
  • 9.4.6.3. Nonprescription topicals,
  • 9.4.6.4. Antimicrobials,
  • 9.4.6.5.Combination topicals,
  • 9.4.6.6. Anti-inflammatories,
  • 9.4.6.7. Hormone blockers,
  • 9.4.6.8. Procedural therapies,
  • 9.5. Discussion,
  • 9.6. Summary and conclusion,
  • 10.1. Lifestyle choices and the acnes,
  • 10.1.1. The "processed cheese queen",
  • 10.2. Therapeutic choices and the acnes,
  • 10.2.1. Acne vulgaris,
  • 10.2.2. Acne rosacea,
  • 10.2.3. Acne inversa/hidradenitis suppurativa,
  • 10.3. Conclusion,
  • 11.1. Appendix A: the rosacea "classification and staging" controversy,
  • 11.2. Appendix B: the dairy versus carbohydrate controversy,
  • 12.1. Acne,
  • 12.2. The "zero-dairy" diet,
  • 12.3. The risks and benefits of isotretinoin,
  • 12.4. The Paleo diet,
  • 12.5. Acne inversa/Hidradenitis suppurativa (AI/HS),
  • 12.6. Yasmin/Ocella/Zarah or Yaz/Gianvi extended cycle for acne therapy.

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