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|b M677c 2009
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|a UAMI
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1 |
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|a Mirzayans, Razmik.
|
| 245 |
1 |
0 |
|a Cellular senescence :
|b implications for cancer therapy /
|c Razmik Mirzayans and David Murray.
|
| 264 |
|
1 |
|a New York :
|b Nova Science Publications, Inc,
|c ©2009.
|
| 300 |
|
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|a 1 online resource
|
| 336 |
|
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|a text
|b txt
|2 rdacontent
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| 337 |
|
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|a computer
|b c
|2 rdamedia
|
| 338 |
|
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|a online resource
|b cr
|2 rdacarrier
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| 504 |
|
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|a Includes bibliographical references (pages 75-100) and index.
|
| 588 |
0 |
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|a Print version record and CIP data provided by publisher.
|
| 505 |
0 |
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|a CELLULAR SENESCENCE:IMPLICATIONS FORCANCER THERAPY; CONTENTS; PREFACE; INTRODUCTION; TELOMERASE-BASED SENESCENCE; STRESS-TRIGGERED PREMATURESENESCENCE; FEATURES OF PREMATURE SENESCENCE; CELL MORPHOLOGY; SENESCENCE-ASSOCIATED Ý-GALACTOSIDASEEXPRESSION; SUSTAINED P21 UP-REGULATION; INHIBITION OF CELL GROWTH; NUCLEAR ACCUMULATION OF ACTIN; STRESS-ASSOCIATED NUCLEAR FOCI; NUCLEAR ACCUMULATION OF ACTIN; STRESS-ASSOCIATED NUCLEAR FOCI; DIFFERENT MODES OF GROWTHARREST ARE TRIGGERED BYGENOTOXIC STRESS; CLASSIFICATION OF CDK INHIBITORS; ROLES OF P21, P53 AND P16IN SENESCENCE.
|
| 505 |
8 |
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|a REGULATION OF P21WAF1 TRANSCRIPTAND P21 PROTEIN LEVELSMODE OF ACTION OF P21; P53 SIGNALING AND CELLULARRESPONSE TO DNA-DAMAGING AGENTS; IONIZING RADIATION-TRIGGERED RESPONSES; UV-TRIGGERED RESPONSES; APOPTOTIC THRESHOLD IN CANCER CELLS EXPOSEDTO DNA-DAMAGING AGENTS; WHAT FACTORS MIGHT CONTRIBUTE TO THEAPOPTOTIC THRESHOLD?; CELL-TO-CELL COMMUNICATIONTRIGGERED BY DNA-DAMAGING AGENTS; ROS-ASSOCIATED BYSTANDER EFFECT OFRADIATION; P53-MEDIATED BYSTANDER EFFECT OF RADIATION; DIFFUSIBLE "AT-CORRECTING" FACTORS; MODULATING SENESCENCE IN THECONTEXT OF CANCER THERAPY.
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| 505 |
8 |
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|a PROS AND CONS OF ACTIVATING SENESCENCE INCANCER CELLSASSAYS FOR PRECLINICAL TESTING OF ANTICANCERAGENTS; AGING, SENESCENCE, AND "HORMESIS"; HUMAN GENETIC DISORDERSASSOCIATED WITH GENOMICINSTABILITY AND CANCERPREDISPOSITION; WERNER SYNDROME; HUTCHINSON-GILFORD'S PROGERIA SYNDROME; BLOOM SYNDROME; ROTHMUND-THOMPSON SYNDROME; COCKAYNE SYNDROME; XERODERMA PIGMENTOSUM; FANCONI ANEMIA; ATAXIA TELANGIECTASIA; LI-FRAUMENI SYNDROME.
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| 505 |
8 |
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|a BIOLOGICAL CONSEQUENCES OF THE FAILURE OF CELLS TO UNDERGO SENESCENCE/APOPTOSIS FOLLOWING GENOTOXIC STRESS: BASIS FOR THE EMERGENCE OF HIGHLY METASTATIC AND THERAPY-RESISTANT DISEASE?CONCLUSION; ACKNOWLEDGMENTS; REFERENCES; INDEX.
|
| 546 |
|
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|a English.
|
| 590 |
|
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|a ProQuest Ebook Central
|b Ebook Central Academic Complete
|
| 650 |
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0 |
|a Cells
|x Aging.
|
| 650 |
|
0 |
|a Cancer
|x Treatment.
|
| 650 |
1 |
2 |
|a Cellular Senescence
|x physiology
|
| 650 |
2 |
2 |
|a Neoplasms
|x therapy
|
| 650 |
|
2 |
|a Cellular Senescence.
|
| 650 |
|
6 |
|a Cellules
|x Vieillissement.
|
| 650 |
|
6 |
|a Cancer
|x Traitement.
|
| 650 |
|
7 |
|a MEDICAL
|x Oncology.
|2 bisacsh
|
| 650 |
|
7 |
|a HEALTH & FITNESS
|x Diseases
|x Cancer.
|2 bisacsh
|
| 650 |
|
7 |
|a Cancer
|x Treatment
|2 fast
|
| 650 |
|
7 |
|a Cells
|x Aging
|2 fast
|
| 700 |
1 |
|
|a Murray, David,
|c Ph. D.
|
| 758 |
|
|
|i has work:
|a Cellular senescence (Text)
|1 https://id.oclc.org/worldcat/entity/E39PCH8xCBVfrhjqWk8PTj6jbq
|4 https://id.oclc.org/worldcat/ontology/hasWork
|
| 776 |
0 |
8 |
|i Print version:
|t Cellular senescence.
|d New York : Nova Biomedical Books, ©2009
|z 1606926764
|w (DLC) 2009499117
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|u https://ebookcentral.uam.elogim.com/lib/uam-ebooks/detail.action?docID=3020128
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