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20240329122006.0 |
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m o d |
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cr cnu|||unuuu |
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050225s1999 dcua ob 100 0 eng d |
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|a 923270139
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|a 0309569338
|q (electronic bk.)
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|a 9780309569330
|q (electronic bk.)
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|a RB127
|b .P37 1999eb
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|2 22
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|a UAMI
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|a National Academy of Sciences Colloquium on the Neurobiology of Pain
|d (1998 :
|c Irvine, Calif.)
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|a Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain /
|c [organized by John Liebeskind, Ronald Dubner and Michael Gold].
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|a Neurobiology of pain
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|a Washington, D.C. :
|b National Academy of Sciences,
|c [1999]
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| 300 |
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|a 1 online resource (pages 7627-7755) :
|b illustrations
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|a text
|b txt
|2 rdacontent
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|a computer
|b c
|2 rdamedia
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|a online resource
|b cr
|2 rdacarrier
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|a National Academy of Sciences colloquium series
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|a " ... papers, which were presented at the National Academy of Sciences colloquium "The Neurobiology of Pain," held December 11-13, 1998, at the Arnold and Mabel Beckman Center in Irvine, CA"--Page 7627.
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|a "July 1999"--Page 7627.
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|a Includes bibliographical references.
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|a Print version record.
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|a COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935�1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS
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| 505 |
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|a SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELSSODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of ... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS?
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| 505 |
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|a CONCLUSIONSCalcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY
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| 505 |
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|a FUNCTIONAL MRI STUDIES IN MONKEYSThe spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY -- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS
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| 505 |
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|a Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptorsCONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord
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| 590 |
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|a ProQuest Ebook Central
|b Ebook Central Academic Complete
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| 590 |
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|a eBooks on EBSCOhost
|b EBSCO eBook Subscription Academic Collection - Worldwide
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| 650 |
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0 |
|a Neurobiology
|v Congresses.
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| 650 |
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0 |
|a Nociceptors
|v Congresses.
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| 650 |
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|a Pain perception
|v Congresses.
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| 650 |
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|a Sodium channels
|v Congresses.
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| 650 |
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0 |
|a Ion channels
|v Congresses.
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| 650 |
|
6 |
|a Neurobiologie
|v Congrès.
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| 650 |
|
6 |
|a Nocicepteurs
|v Congrès.
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| 650 |
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6 |
|a Perception de la douleur
|v Congrès.
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| 650 |
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6 |
|a Canaux à sodium
|v Congrès.
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| 650 |
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6 |
|a Canaux ioniques
|v Congrès.
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| 650 |
|
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|a HEALTH & FITNESS
|x Pain Management.
|2 bisacsh
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| 650 |
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|a MEDICAL
|x Pain Medicine.
|2 bisacsh
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| 650 |
|
7 |
|a Ion channels
|2 fast
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| 650 |
|
7 |
|a Neurobiology
|2 fast
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| 650 |
|
7 |
|a Nociceptors
|2 fast
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| 650 |
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|a Pain perception
|2 fast
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| 650 |
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|a Sodium channels
|2 fast
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| 655 |
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|a Conference papers and proceedings
|2 fast
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| 700 |
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|a Liebeskind, John C.
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|a Dubner, Ronald.
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|a Gold, Michael Robert.
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|a National Academy of Sciences (U.S.)
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0 |
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|a Proceedings of the National Academy of Sciences of the United States of America.
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|a Colloquium on the Neurobiology of Pain.
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|a Colloquium on Neurobiology of Pain.
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|i Print version:
|a National Academy of Sciences Colloquium on the Neurobiology of Pain (1998 : Irvine, Calif.).
|t Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain.
|d Washington, D.C. : National Academy of Sciences, [1999]
|w (OCoLC)54038127
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| 830 |
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|a National Academy of Sciences colloquium series.
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| 856 |
4 |
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|u https://ebookcentral.uam.elogim.com/lib/uam-ebooks/detail.action?docID=3377386
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