Copper in drinking water /
Clasificación: | Libro Electrónico |
---|---|
Autor Corporativo: | |
Formato: | Electrónico eBook |
Idioma: | Inglés |
Publicado: |
Washington, D.C. :
National Academy Press,
©2000.
|
Temas: | |
Acceso en línea: | Texto completo |
Tabla de Contenidos:
- Copper in Drinking Water
- Copyright
- Preface
- Contents
- Executive Summary
- THE CHARGE TO THE COMMITTEE
- THE COMMITTEE'S APPROACH TO ITS CHARGE
- THE COMMITTEE'S EVALUATION
- Health Effects of Excess Copper
- Recommendations
- Physiological Role of Copper
- Recommendations
- Sensitive Populations
- Recommendations
- Risk Characterization
- Recommendations
- 1 Introduction
- CHEMICAL AND PHYSICAL PROPERTIES
- SOURCES OF COPPER IN DRINKING WATER
- COMMITTEE'S APPROACH TO ITS CHARGE
- STRUCTURE OF THE REPORT
- REFERENCES
- 2 Physiological Role of CopperESSENTIALITY
- BIOCHEMISTRY AND PHYSIOLOGY
- FACTORS AFFECTING BIOAVAILABILITY
- Effect of Age
- Dietary and Other Interactions
- CONCLUSIONS
- RECOMMENDATIONS
- REFERENCES
- 3 Health Effects of Copper Deficiencies
- TERATOGENESIS OF COPPER DEFICIENCY
- Causes of Copper Deficiency
- Copper in Prenatal Development
- Drug-Induced Copper Deficiency
- Disease-Induced Copper Deficiency
- Copper-Diet Interactions
- Gene-Induced Copper Deficiency
- Human Copper Deficiency and Teratogenesis
- Primary Dietary Copper DeficiencyHEALTH EFFECTS OF COPPER DEFICIENCIES IN ADULTS
- CONCLUSIONS
- REFERENCES
- 4 Disorders of Copper Homeostasis
- MENKES DISEASE
- OCCIPITAL HORN SYNDROME
- WILSON DISEASE
- GENETIC CHARACTERISTICS OF WILSON AND MENKES DISEASES
- HETEROZYGOTES FOR WILSON DISEASE
- ACERULOPLASMINEMIA
- TYROLEAN INFANTILE CIRRHOSIS
- INDIAN CHILDHOOD CIRRHOSIS
- IDIOPATHIC COPPER TOXICOSIS
- OTHER GENETIC DISORDERS
- DISEASE-INDUCED CHANGES IN COPPER HOMEOSTASIS
- CONCLUSIONS
- RECOMMENDATIONS
- REFERENCES
- 5 Health Effects of Excess CopperACUTE TOXICITY
- Case Reports and Population-Based Studies
- CHRONIC TOXICITY
- Reproductive and Developmental Toxicity
- Genotoxicity, Mutagenicity, and Carcinogenicity of Copper
- Sensitive Populations
- Carriers of Genetic Defects in Copper Homeostasis
- Glucose-6-Phosphate Dehydrogenase Deficiency (G6PD)
- ANIMAL STUDIES
- Toxicity in Animals
- Reproductive and Developmental Toxicity
- Neurotoxicity
- Carcinogenicity
- Mechanisms and Animal Models for Copper Toxicity
- Mechanism of Acute Copper Toxicity
- Mechanism of Chronic Copper ToxicityCONCLUSIONS
- RECOMMENDATIONS
- REFERENCES
- 6 Risk Characterization
- COPPER DEFICIENCY
- COPPER TOXICITY FROM SINGLE OR SHORT-TERM EXPOSURE
- COPPER TOXICITY FROM CHRONIC EXPOSURE
- Sensitive Populations
- Carriers of the Wilson-Disease Gene and Other Genetically Sensitive Groups
- Infants
- Chronic Liver Disease
- Glucose-6-Phosphate Dehydrogenase Deficiency
- Implications for the MCLG
- CHRONIC COPPER EXPOSURE THROUGH TAP WATER
- DIETARY CONTRIBUTION AND TOTAL COPPER INTAKE
- Formula-Fed Infants