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978-3-319-32337-4 |
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|a 9783319323374
|9 978-3-319-32337-4
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|a 10.1007/978-3-319-32337-4
|2 doi
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|a RC321-580
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|a 612.8
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|a Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke
|h [electronic resource] /
|c edited by Jun Chen, John H. Zhang, Xiaoming Hu.
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|a 1st ed. 2016.
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|a Cham :
|b Springer International Publishing :
|b Imprint: Springer,
|c 2016.
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|a XIII, 408 p. 49 illus., 44 illus. in color.
|b online resource.
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|a text
|b txt
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|a computer
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|a online resource
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|a Springer Series in Translational Stroke Research,
|x 2363-9598
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|a Part 1. Microvascular integrity in stroke -- 1. Structural alterations to the endothelial tight junction complex during stroke -- 2. Role of pericytes in neurovascular unite and stroke -- 3. Glial support of blood-brain barrier integrity: Molecular targets for novel therapeutic strategies in stroke -- 4. Barrier mechanism in neonatal stroke -- 5. Angiogenesis: a realistic therapy for ischemic stroke -- Part 2. Glial cells in stroke -- 6. Astrocytes as a target for ischemic stroke -- 7. Microglia: a double-sided sword in stroke -- 8. Crosstalk between cerebral endothelium and oligodendrocyte after stroke -- Part 3. Peripheral immune cells in stroke -- 9. The peripheral immune response to stroke -- 10. The Role of spleen-derived immune cells in ischemic brain injury -- 11. Regulatory T cells in ischemic brain injury -- 12. B cells in stroke and preconditioning-induced protection against stroke -- 13. Mast cell as an early responder in ischemic brain injury -- 14. Roles of neutrophils in stroke -- 15. The function of cytokines in ischemic stroke -- Part 4. White matter injury and repair in stroke -- 16. Ischemic injury to white matter: an age-dependent process -- 17. Neurovascular repair after stroke -- 18. The role of non-neuronal Nrf2 pathway in ischemic stroke: damage control and potential tissue repair -- 19. Stem cell therapy for ischemic stroke.
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|a This book provides a comprehensive overview of the latest research in the role of non-neuronal cells - astrocytes, oligodendrocytes, endothelial cells, pericytes, microglia, and other immune cells in ischemic brain injury and long-term recovery. In these cases, neurodegeneration and brain repair are controlled in a sophisticated system, incorporating interactions between different cell types and cellular systems. Also explored are the therapeutic strategies that target non-neuronal responses after stroke and their translational potentials.
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|a Neurosciences.
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|a Neurology .
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|a Neuroscience.
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|a Neurology.
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|a Chen, Jun.
|e editor.
|4 edt
|4 http://id.loc.gov/vocabulary/relators/edt
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700 |
1 |
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|a Zhang, John H.
|e editor.
|4 edt
|4 http://id.loc.gov/vocabulary/relators/edt
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700 |
1 |
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|a Hu, Xiaoming.
|e editor.
|4 edt
|4 http://id.loc.gov/vocabulary/relators/edt
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710 |
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|a SpringerLink (Online service)
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773 |
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|t Springer Nature eBook
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|i Printed edition:
|z 9783319323350
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776 |
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|i Printed edition:
|z 9783319323367
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|i Printed edition:
|z 9783319812601
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830 |
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|a Springer Series in Translational Stroke Research,
|x 2363-9598
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4 |
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|u https://doi.uam.elogim.com/10.1007/978-3-319-32337-4
|z Texto Completo
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|a ZDB-2-SME
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|a ZDB-2-SXM
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|a Medicine (SpringerNature-11650)
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950 |
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|a Medicine (R0) (SpringerNature-43714)
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