Pathological Potential of Neuroglia Possible New Targets for Medical Intervention /
Pathophysiological states, neurological and psychiatric diseases are almost universally considered from the neurocentric point of view, with neurons being the principal cellular element of pathological process. The brain homeostasis, which lies at the fulcrum of healthy brain function, the compromis...
Clasificación: | Libro Electrónico |
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Autor Corporativo: | |
Otros Autores: | , |
Formato: | Electrónico eBook |
Idioma: | Inglés |
Publicado: |
New York, NY :
Springer New York : Imprint: Springer,
2014.
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Edición: | 1st ed. 2014. |
Temas: | |
Acceso en línea: | Texto Completo |
Tabla de Contenidos:
- General Pathophysiology of Neuroglia: Neurological and Psychiatric Disorders as Gliopathies
- Ionic Signaling in Physiology and Pathophysiology of Astroglia
- Pathophysiology of Vesicle Dynamics in Astrocytes
- Glial Glutamate and Metabolic Transporters as a Target for Neurodegenerative Therapy and Biomarkers
- Alexander Disease and Astrotherapeutics
- Role of Astrocytes in Central Nervous System Traum
- Astroglial and Neuronal Integrity During Cortical Spreading Depolarization
- Crucial role for astrocytes in epilepsy
- Microglial Biology in Neuroinflammatory Disease: Pharmaco-industrial Approach to Target Validation
- The Role of Astrocytes in Huntington's Disease
- Amyotrophic Lateral Sclerosis: A Glial Perspective
- Complex and Associated Astroglial and Microglial Changes during the Progression of Alzheimer´s Disease
- Possible Therapeutic Targets in Microglia
- Novel Therapeutic Approaches to Malignant Gliomas
- Hepatic encephalopathy: A Primary Neurogliopathy
- Astroglia and Severe Mental Illness: A Role for Glutamate Microdomains
- Emerging Roles for Glial Pathology in Addiction
- Astroglial Connexins as Elements of Sleep-wake Cycle Regulation and Dysfunction
- Experimental Treatment of Acquired and Inherited Neuropathies
- Satellite glial cells as target for chronic pain therapy
- Enteric Glial Cells: Implications in Gut Pathology.