Molecular Defects in Cardiovascular Disease
Molecular Defects in Cardiovascular Disease provides an in-depth discussion of the molecular mechanisms underlying the genesis of cardiovascular defects and the implications this has on current and emerging targeted therapeutics. Divided into three sections, this book covers the scientific foundati...
Clasificación: | Libro Electrónico |
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Autor Corporativo: | |
Otros Autores: | , , |
Formato: | Electrónico eBook |
Idioma: | Inglés |
Publicado: |
New York, NY :
Springer New York : Imprint: Springer,
2011.
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Edición: | 1st ed. 2011. |
Temas: | |
Acceso en línea: | Texto Completo |
Tabla de Contenidos:
- Calcium overloading-induced oxidative stress-mediated cellular and subcellular remodeling
- Intracellular MMP-2: Role in normal and diseased hearts
- Control of the mesenchymal-derived cell phenotype by Ski and Meox2: A putative mechanism for post-developmental phenoconversion
- Elevated Na+/H+ exchanger expression and its role in myocardial disease.- Impact of perinatal chronic hypoxia on cardiac tolerance to acute ischemia
- Stem cell, microRNA and redox cycling
- Cardiac cell therapy: Present and future
- Macroscopic and microscopic aspects of cardiac dysfunction in congestive heart failure
- Cardiac dysfunction and metabolism: Unravelling the molecular cross-talk
- Metabolic remodeling of the hypertrophied heart
- Mechanisms for the regulation of phospholipase C gene expression in cardiac hypertrophy
- Molecular changes in fatty acid oxidation in the failing heart
- Cardiolipin metabolism in experimental and human heart failure
- Mechanisms underlying development of cardiomyocyte hypertrophy via Na-H exchange stimulation
- The structural basis of cardiac dysfunction in human heart failure
- Hormonal mechanisms of cardiac remodeling in heart failure
- Modulators of remodeling after myocardial infarction
- Phosphodiesterase-5 inhibitors in protection against doxorubicin-induced cardiomyopathy
- Molecular basis of pulmonary hypertension in left heart failure
- Modulation of Gi protein expression in hypertension: Molecular mechanisms
- Vascular dysfunction in heart disease
- Cellular and molecular mechanisms associated with salicylate inhibition of intimal hyperplasia balloon catheter-induced vascular injury
- Involvement of growth factor receptor and non receptor protein tyrosine kinases in endothelin 1 and angiotensin II-induced signaling pathways in the cardiovascular system
- Sarco/endoplasmic reticulum Ca2+ pump damage by oxidative stress: Implications for Ca2+ entry
- Obesity-hypertension: Leptin as the common link to cardiovascular and renal dysregulation
- Cardiorenal protection in diabetes mellitus
- Molecular mechanisms in the pathogenesis of diabetic cardiomyopathy.