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Neurochemical Aspects of Excitotoxicity
About 40% of central nervous system synapses use glutamate as the neurotransmitter. Over-stimulation of glutamate receptors produces neuronal injury or death by excitotoxicity, which is closely associated with neurochemical and neuropathological changes involved in acute neural trauma (stroke, spina...
| Clasificación: | Libro Electrónico |
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| Autores principales: | , , |
| Autor Corporativo: | |
| Formato: | Electrónico eBook |
| Idioma: | Inglés |
| Publicado: |
New York, NY :
Springer New York : Imprint: Springer,
2008.
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| Edición: | 1st ed. 2008. |
| Temas: | |
| Acceso en línea: | Texto Completo |
Tabla de Contenidos:
- Glutamate and Aspartate in Brain
- Excitatory Amino Acid Receptors in Brain
- Multiplicity of Glutamate Receptors in Brain
- Glutamate Transporters and Their Role in Brain
- Excitatory Amino Acid Receptors and Their Association with Neural Membrane Glycerophospholipid Metabolism
- Glutamate Receptors and Their Association with Other Neurochemical Parameters in Excitotoxicity
- Possible Mechanisms of Neural Injury Caused by Glutamate and Its Receptors
- Glutamate Receptors and Neurological Disorders
- Endogenous Antioxidant Mechanisms and Glutamate Neurotoxicity
- Glutamate Receptor Antagonists and the Treatment of Neurological Disorders
- Future Perspectives: New Strategies for Antagonism of Excitotoxicity, Oxidative Stress and Neuroinflammation in Neurodegenerative Diseases.